Femoroacetabular impingement (FAI) is recognized as a pathomechanical process that leads to hip osteoarthritis (OA) [1-3]. It is hypothesized that mechanical stimuli are more prominent in an impinged joint at a higher range of motion. Adverse loading conditions can impose elevated mechanical stimuli levels at the articulating surfaces and underlying subchondral bone, which has a predominant mechanical role in early OA [4]. Despite past attempts to characterize mechanisms by which cam FAI (aspherical femoral head-neck deformity) leads to OA, very little has been done to integrate clinical patient-specific data to determine stresses and strains within the symptomatic hip joint. The aim of this research is to determine the levels of mechanical stimuli within the hip, examining the effects of severe cam FAI on the onset of OA, using patient-specific biomechanics data, CT data, and finite element analysis (FEA).